Read e-book online Aluminum and renal failure PDF

By Marc E. de Broe, Patrick C. D’Haese (auth.), Marc E. de Broe, Jack W. Coburn (eds.)

ISBN-10: 0792303474

ISBN-13: 9780792303473

ISBN-10: 9400918682

ISBN-13: 9789400918689

ISBN-10: 9401073333

ISBN-13: 9789401073332

` ... the fabric awarded presents useful details in regards to the organic results and scientific syndromes linked to aluminum publicity. This quantity will be priceless for college students and nephrologists who're attracted to furthering their realizing of the area.'
Dialysis and Transplantation, 20/1, January 1991

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Extra resources for Aluminum and renal failure

Example text

706 V) [49], anions are strongly attracted by aluminum. 000 mg/doy Figure 1. Metabolism of aluminum in the presence of (left) a normal renal function and (right) end-stage renal failure. fonning relatively stable salts which do not ionize readily. The chemical nature of aluminum in water is essentially the chemistry of AI(OH)3 which has an amphoteric character and a tendency to form complex ions and to polymerize. Thus, the form and concentration of aluminum in water depends on the pH and the nature of accompanying substances.

In an attempt to reverse this process, Biswas et al. [29] studied PTH levels in 20 patients during Al hydroxide treatment to bind intestinal phosphate. In spite of decrease in serum phosphate and a concomitant rise in serum calcium, the PTH levels remained to be high, whereas serum Al rose significantly. In later studies, reviewed by Slatopolsky [30], decreased PTH levels were observed in patients with Al accumulation. It is as yet unclear whether this is a result of a direct effect of Al on the parathyroid glands, or because serum calcium concentrations are relatively high in this group of patients.

To solve this interindividual variation, research has to be performed into the mechanism of Al transport through the intestinal mucosa. This subject is dealt with in Section 4. 44 Aluminum absorption in non-renal patients Data on Al absorption in other than renal patients are scarce so far. Williams et ai. [19] showed that Al disposition in bone appeared in 2 patients with hepatic disease but with normal renal function. Both had been on long-term antacid therapy. Impairment of biliary Al excretion was proposed as the cause of Al accumulation in liver failure.

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Aluminum and renal failure by Marc E. de Broe, Patrick C. D’Haese (auth.), Marc E. de Broe, Jack W. Coburn (eds.)

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